Results: The authors identified and replicated 28 independent GRNs active in CAD.

The antibody titer in serum was determined by ELISA using biotin-conjugated GRN.

Several important genes were revealed based on the rewiring of the reconstructed GRNs.

Deep sequencing of families exhibiting late-onset dementia revealed several novel variants in GRN.

Here, we use computational models of GRNs to investigate this relationship.

Dynamic GRNs at four different stages of colorectal cancer were reconstructed and analyzed.

The same method can potentially be used to find homeostasis regions in other GRNs.

In support of this hypothesis, we show that the feed-forward loop GRN produces homeostasis.

How assortativity influences the robustness of the signal-integration logic of GRNs remains an open question.

This instrument is supplemented by a set of cognitive tests, prospectively validated in the GRNS.

Gene regulatory networks (GRNs) drive the cellular processes that sustain life.

Screening for GRN mutations should also be considered in CBS patients without a positive family history.

This finding opens avenues for future research into GRN biology and the pathophysiology of neurodegenerative diseases.

Our data provide basic molecular information useful for the further investigation on the function of GRN gene.

This study also showed a wide range in plasma GRN levels in non-GRN mutation carriers, including controls.

We previously hypothesized that a neural crest gene regulatory network (NC-GRN) guides neural crest formation.